Spinal anesthesia (SA) is a safe anesthetic technique used for both elective and emergency operations. Shivering is known to be a frequent complication in patients undergoing surgery under neuraxial anesthesia with incidence of 40–70%. SA inhibits tonic vasoconstriction and causes redistribution of core heat from the trunk (below the block level) to the peripheral tissues predisposing patients to hypothermia and shivering. Post spinal anaesthesia shivering (PSAS) is an involuntary, repetitive activity of skeletal muscles as a physiological response to core hypothermia to raise the metabolic heat production. PSAS increases O2 consumption, CO2 production, plasma catecholamines and cardiac output. Shivering may interfere with the monitoring of ECG, blood pressure and oxygen saturation. Te mainstay of prophylaxis and treatment of PSAS remain pharmacological due to inadequate control of central hypothermia by techniques based on physical principles (e.g., intravenous infusion (IVI) of warm fluids and forced air warmers). It appears logical to prevent PSAS rather than to treat it once it develops.